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OI not simply suppressed lipopolysaccharide-induced proinflammatory cascades of inducible nitric oxide synthase, cyclooxygenase-2, and cytokines release in mouse BV2 microglial cells but also activated the Nrf2 signaling pathway and its downstream targets in these cells. Conditioned medium derived from OI-addressed BV2 cells protected towards roten

Following, to decipher the mechanism by which LDHA modulates the expression of IκB-ζ and also the inflammatory reaction, we 1st established that LDHA doesn't control mRNA balance of Nfkbiz, suggesting that IκB-ζ is probably going regulated at or further than the phase of translation (Supplementary Fig. S4A). This is often in settlement with ear